In published research in the Journal of Restorative Medicine, compelling evidence is shown that acetaminophen (sold as Tylenol or Paracetamol) (does not cause autism) but increases the incidence of autism, attention deficit with hyperactivity and asthma in genetically and/or metabolically susceptible children. In the US, acetaminophen is sold over-the-counter, in spite of the fact that it results in death for hundreds of Americans each year.
The study compares autism rates in the US with Cuba, where acetaminophen is only available via prescription, and not widely used. The percentage of the population with autism in the United States is 298 times higher than in Cuba.
[AND FOR THE RECORD, WHAT YOU WILL BE READING DOES NOT LET VACCINES OFF THE HOOK ONE BIT, BECAUSE THEY CONTAIN BRAIN-DAMAGING ALUMINUM, UNAVOIDABLE BACTERIOPHAGE, AS WELL AS THE VIRUSES AND CONTAMINANTS THAT CAN CAUSE SIGNIFICANT INJURY (SEE FULL LIST) TO A CHILD OR ADULT. THESE INJURIES INCLUDE BRAIN SWELLING AND PERMANENT BRAIN INJURY, SEIZURES AND CONVULSIONS, AND EVEN DEATH. SINCE 1988 WELL OVER 3 BILLION DOLLARS IN COMPENSATION HAS BEEN PAID BY THE FEDERAL GOVERNMENT TO VACCINE VICTIMS. ]
It makes absolutely no sense … at first …until we look at what happens in the body.
It all starts with glutathione. (I’m summarizing this, OK?)
“Oxidation” happens in the body when cells make energy – which is all the time – especially your brain and liver which never stop working. Harmful unstable molecules called “free radicals” are created as a byproduct. Free radicals are “bad” because they can damage cell DNA, potentially causing permanent mutations. The body uses antioxidants to “scavenge” free radicals, neutralizing them so they can’t damage the DNA or any other part of the cell.
Glutathione is the body’s most important self-made antioxidant. We also get some antioxidants from foods: pomegranates, dark chocolate and berries, for example.
(Bear with me, I’m getting to the point soon, I promise.)
Glutathione can absorb the impact of many of these normal free radicals, but when the body takes a large oxidative “hit” (like ingredients in vaccines and other constant environmental toxins added together) it causes the depletion of active glutathione. You might say it is all used up – there is not enough to go around cleaning up. The result is lots and lots and lots of oxidative damage – especially in the liver and the brain.
Glutathione is especially important because it is highly active in the brain, unlike most other antioxidants. Imagine what would happen if you deplete glutathione in the brain. You end up with oxidative damage, inflammation and sometimes permanent brain injury.
And is there a pharmaceutical known for depleting glutathione? Unfortunately, yes! It’s acetaminophen (Tylenol).
A number of scientists (including prestigious JAMA Pediatrics) have independently published molecular mechanisms by which acetaminophen can induce inflammation [1, 2, 3, 4, 5]. The drug can profoundly alter toxin metabolism and inflammatory processes in the body. It actually makes sense that acetaminophen causes brain damage in some children.
Acetaminophen is the last remaining drug in a particular class of relatively toxic drugs that is derived from coal tar. It is the only drug left in its class not discontinued because of toxicity. It was already known to be associated with the development of asthma in children [6, 7]. Oddly enough, it was not known if acetaminophen actually worked to alleviate fever in children .
Dr. Erica Krumbeck explains this way better than I could:
Tylenol is actually somewhat famous for depleting glutathione . Tylenol overdoses (which occur at remarkably low doses compared to other pharmaceuticals, by the way) cause massive liver damage. The reason is the way it’s processed in the liver. The toxic chemical Tylenol is broken down into requires a HUGE amount of glutathione to make it less toxic – and it is used up. Acetaminophen, actually, is the #1 cause of liver failure in the U.K., and the second most common cause in the U.S.
It makes a lot of sense, actually, given what we know about oxidative damage in the brain. Many children with ASD have poor transsulfuration and methylation – they can’t make glutathione and even worse, they can’t activate many neurotransmitters in the brain. Remember that glutathione is one of few compounds that can cross the blood-brain barrier which makes it extremely important in the prevention of neurological disorders.
So this is why Tylenol could possibly trigger autism in kids who are genetically susceptible. Please understand me on this one – not every child who gets Tylenol will get autism. And not every child who has the gene and gets Tylenol will get autism. I believe that developing ASD is a combination of genes and environmental insult – basically you must be genetically predisposed and then be exposed to a multitude of things that deplete glutathione. (source)
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